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Tamoxifen may cause damage to developing embryos. You should use an effective non-hormonal type of birth control ‘ such as condoms, a diaphragm along with spermicide, or a non-hormonal I. Ask your doctor which type of non-hormonal birth control would be best for you. Since its approval in 1998, tamoxifen has been used to treat millions of women and men diagnosed with hormone-receptor-positive breast cancer, bold 300 mg injectable steroids equipoise, eq. While an aromatase inhibitor is the first hormonal therapy medicine choice for postmenopausal women, tamoxifen is the first choice for premenopausal women and is still a good choice for postmenopausal women who can’t take an aromatase inhibitor. Tamoxifen offers other health benefits that aren’t related to treating cancer. Because it’s a SERM, it selectively either blocks or activates estrogen’s action on specific cells. While tamoxifen blocks estrogen’s action on breast cells, it activates estrogen’s action in bone and liver cells. So tamoxifen can: help stop bone loss after menopause lower cholesterol levels. Reasons some people may not get the full benefit of tamoxifen. The body uses an enzyme called CYP2D6 to convert tamoxifen into its active form. Two things can interfere with the body’s ability to make this happen: a flaw in the CYP2D6 enzyme and certain medications that block the effectiveness of this enzyme. Abnormal CYP2D6 enzyme: About 10% of people have a CYP2D6 enzyme that doesn’t function as well as it should. Having an abnormal CYP2D6 enzyme might keep a person from getting the full benefit of tamoxifen. You may want to ask your doctor about being tested for this enzyme abnormality if you are considering taking tamoxifen. Still, CYP2D6 testing is controversial because several large studies found that an abnormal CYP2D6 enzyme didn’t affect tamoxifen’s effectiveness. Together, you and your doctor can decide if CYP2D6 testing makes sense for your unique situation. Medications that can interfere with CYP2D6: There are medications that can block the activity of CYP2D6 to varying degrees (usually stated as ‘strong’ or ‘moderate’ inhibitors of CYP2D6). These medications include some of the antidepressants known as selective serotonin reuptake inhibitors (SSRIs) and serotonin-norepinephrine reuptake inhibitors (SNRIs). There are also other types of commonly prescribed medications, such as Cardioquin (chemical name: quinidine), Benadryl (chemical name: diphenhydramine), and Tagamet (chemical name: cimetidine), that can block CYP2D6. Blocking the activity of CYP2D6 can interfere with the activation of tamoxifen ‘ reducing its effectiveness as an anti-cancer treatment. Most doctors recommend that you avoid taking strong and moderate inhibitors of CYP2D6 while you’re on tamoxifen. If you have already finished tamoxifen and you were taking other medications at the same time, make an appointment to talk with your doctor about whether any of your other medications may have interfered with CYP2D6 and the potential benefit you received from tamoxifen. Your doctor may recommend no additional therapy or extended hormonal therapy (with tamoxifen or an aromatase inhibitor) depending on your risk of recurrence, your overall medical condition, and your preferences. If you were taking tamoxifen because you are at high risk but have never been diagnosed, and you were also taking a CYP2D6 inhibitor, your doctor may now recommend additional anti-estrogen therapy with tamoxifen or raloxifene (brand name: Evista), depending on your menopausal status. undefined — bold 300® is an anabolic steroid possessing anabolic properties with moderate androgenic activity. Anabolic and androgenic agents are widely. Deca durabolin o boldenone – bold eq 300 mg. 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